Around the world over thousands of years, patients have received root-cause holistic treatment for their diseases with personalized
treatment, diet and lifestyle modification recommendations. Read the inspiring true stories of practitioners who heal people and who recovered
from their problems after adhd-add treatment at their clinics. Many have been generous to share their knowledge and experience for the benefit
of other holistic experts and patients alike. Many practitioners share their Case Studies and the healing powers of adhd-add and related therapies
as they heal people who benefited from our expertise.
Thimerosal exposure in infants and neurodevelopmental disorders: an assessment of computerized medical records in the Vaccine Safety Datalink.
August 2008
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/>J Neurol Sci. 2008 Aug 15;271(1-2):110-8. Epub 2008 May 15. PMID: 18482737
Activation of methionine synthase by insulin-like growth factor-1 and dopamine: a target for neurodevelopmental toxins and thimerosal.
March 2004
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/>Mol Psychiatry. 2004 Apr ;9(4):358-70. PMID: 14745455
EFA supplementation in children with inattention, hyperactivity, and other disruptive behaviors.
October 2003
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/>Lipids. 2003 Oct;38(10):1007-21. PMID: 14669965
A double-blind, parallel, multicenter comparison of L-acetylcarnitine with placebo on the attention deficit hyperactivity disorder in fragile X syndrome boys.
April 2008
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/>Am J Med Genet A. 2008 Apr 1;146(7):803-12. PMID: 18286595
Artificial food colouring and hyperactivity symptoms in children.
October 2009
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/>Prescrire Int. 2009 Oct;18(103):215. PMID: 19882794
Potential effects of zinc on information processing in boys with attention deficit hyperactivity disorder.
April 2008
METHODS:
28 boys with ADHD were divided into two groups according to plasma zinc levels: low zinc group (N=13, zinc level <80 microg/dL) and zinc non-deficient group (N=15, zinc level >or=80 microg/dL). ERP indices from parietal and frontal brain regions were recorded in children with ADHD and in 24 normal boys by using an auditory oddball paradigm. Plasma zinc levels were measured by an atomic absorption spectrophotometer.
Results:
The plasma zinc levels were significantly lower in both ADHD groups (means are 65.8 microg/dL in low zinc group and 89.5 microg/dL in zinc non-deficient group) than controls (mean: 107.8 microg/dL; both p values <0.017). In ADHD compared to controls, the amplitudes of P3 in frontal and parietal regions were significantly lower, and the latency of P3 in parietal region was significantly longer (all p values <0.017). In low zinc ADHD group compared to zinc non-deficient ADHD group, the latencies of N2 in frontal and parietal region were significantly shorter (all p values <0.017). In addition, there was a medium but significant positive correlation between plasma zinc levels and amplitude and latency of frontal N2 wave in ADHD. CONCLUSIONS: These results can suggest that plasma zinc levels might have an effect on information processing in ADHD children, and lower zinc levels seem to affect N2 wave. Since N2 wave changes may reflect a different inhibition process, further studies are warranted to investigate the effect of zinc on inhibitory process in children with ADHD, and in low zinc and non-deficient ADHD groups.
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/>Prog Neuropsychopharmacol Biol Psychiatry. 2008 Apr 1;32(3):662-7. Epub 2007 Nov 17. PMID: 18083281